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Download PDF by Hymie Anisman, Steve Zalcman, Nola Shanks (auth.), Alan A.: Animal Models in Psychiatry, II

By Hymie Anisman, Steve Zalcman, Nola Shanks (auth.), Alan A. Boulton, Glen B. Baker, Mathew T. Martin-Iverson (eds.)

ISBN-10: 0896031772

ISBN-13: 9780896031777

ISBN-10: 159259624X

ISBN-13: 9781592596249

The Animal types in Psychiatry volumes are loosely prepared via topic. the 1st quantity features a variety of chapters fascinated with schizophrenia, psyc- ses, neuroleptic-induced tardive dyskinesias, and different d- orders that could contain dopamine, reminiscent of realization deficit affliction and mania. additionally integrated is a bankruptcy describing a behavioral version for activity-induced anorexia. the second one quantity bargains with affective and nervousness problems, but additionally comprises chapters on matters no longer simply labeled as both psychotic, affective, or anxiety-related, corresponding to aggression, psychological retardation, and reminiscence issues. 4 chapters on animal versions of schizophrenia or psychoses are incorporated in quantity 18 a result of significance of those problems in psychiatry. Likewise, 3 chapters within the current v- ume care for affective issues, with a fourth bankruptcy on circadian rhythms that still contributes to tools for a- mal types in affective problems. Following the 1st 4 chapters are chapters facing versions of tension and panic, chapters on aggression, one on psychological retardation, and a last bankruptcy protecting reminiscence problems. a few of the behaviorally-based versions of affective problems contain inducing tension in a- mals, frequently on a prolonged foundation. the 1st bankruptcy by way of Anisman, Zalcman, Shanks, and Zacharko describes a number of the neurochemical results which are linked to the persistent software of sensors.

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Extra resources for Animal Models in Psychiatry, II

Sample text

It will be recognized that the hormonal and neurochemical consequences of antigenic challenge are reminiscent of the effects ordinarily elicited by stressors. Moreover, many of the immunological consequences of stressors are remarkably consistent with the neurochemical effects associated with such a treatment. As a working hypothesis it might be supposed that antigens, possibly through IL-l, act on endogenous processes much in the same way as stressors do (Dunn, 1988), and as such might ultimately engender behavioral effects like those produced by stressors.

As shown in Fig. 8, the escape disturbances induced by inescapable Animal Model of Depression 41 shock in A/J mice was eliminated by repeated treatment with DMI. In contrast, the drug was ineffective in ameliorating the behavioral disturbance in either the BALBlcByJ, C57BL/6J, or CD-l strain. In contrast, repeated bupropion treatment marginally reduced the deficit in the CD-1 mouse, but did not affect performance in the other strains. Finally, repeated treatment with amitriptyline reduced the deficits in A/J and CD-l mice, although acute treatment was also effective in this respect (Shanks and Anisman, 1989).

1985). It will be recognized that the hormonal and neurochemical consequences of antigenic challenge are reminiscent of the effects ordinarily elicited by stressors. Moreover, many of the immunological consequences of stressors are remarkably consistent with the neurochemical effects associated with such a treatment. As a working hypothesis it might be supposed that antigens, possibly through IL-l, act on endogenous processes much in the same way as stressors do (Dunn, 1988), and as such might ultimately engender behavioral effects like those produced by stressors.

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Animal Models in Psychiatry, II by Hymie Anisman, Steve Zalcman, Nola Shanks (auth.), Alan A. Boulton, Glen B. Baker, Mathew T. Martin-Iverson (eds.)


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